sepanjang hari duk doing nonsense. sebab 2 macam xtaw nak watpe jer dalam kelas hari nie. stress??nope, klu tak, da lama kepala aku sakit..bosan??nope, kalau tak, da lama aku tido dari balik kelas tadi...crazy??yes..maybe sebab takder per untuk boost otak aku bagi berfungsi dangan baik...huhuhu.plis givme problem to be solve like yesterday...
"why hyperglycemia on diabetic patient can be COD in animal?, hypo pung mapuh, hyper pung mapuh. dok reti goh la.."
hyperglycemia is the state where the glucose in blood vessel is higher than normal range. when this occur, it will drawn out natrium from cell to the body. sbb 2 glucose blh guner untuk draw in natrium from blood into tthe cell if and only if insulin is normally produced. klu x. natrium plak yang keluar freely to the blood vessel and cause elactrolyte imbalance to cell. klu da terlebih banyak natrium hilang, potassium plak yg terlebih banyak dalam cell.potassium paired ngan calcium, so, it will draw in calcium into cell...so, guess what next??? calcium related to muscle activity. so, hyperexcited la kowt. glucose xle guner, protein jadi mangsa utk provide tenaga. end product for limited time will be ketone bodies that actual kos metabolic acidosis to the system...what next??x mati2 lagi nie...amik potassium jer la, higher potassium, increase cardiac output, lead to circulatory failure when the heart is failed to campensate with the exteremely immediate change..DO in blood kureng, vital organ failure, brain x dpt enough oxygen--> brain cell death, so the host will death also..mati pun host sekor nie...ntah betul ke tak tau aku tulis nie. malas da nak rujuk buku..nanti jer la.. (",)..
end of yesterday. really need problem for today... :-(
"why hyperglycemia on diabetic patient can be COD in animal?, hypo pung mapuh, hyper pung mapuh. dok reti goh la.."
hyperglycemia is the state where the glucose in blood vessel is higher than normal range. when this occur, it will drawn out natrium from cell to the body. sbb 2 glucose blh guner untuk draw in natrium from blood into tthe cell if and only if insulin is normally produced. klu x. natrium plak yang keluar freely to the blood vessel and cause elactrolyte imbalance to cell. klu da terlebih banyak natrium hilang, potassium plak yg terlebih banyak dalam cell.potassium paired ngan calcium, so, it will draw in calcium into cell...so, guess what next??? calcium related to muscle activity. so, hyperexcited la kowt. glucose xle guner, protein jadi mangsa utk provide tenaga. end product for limited time will be ketone bodies that actual kos metabolic acidosis to the system...what next??x mati2 lagi nie...amik potassium jer la, higher potassium, increase cardiac output, lead to circulatory failure when the heart is failed to campensate with the exteremely immediate change..DO in blood kureng, vital organ failure, brain x dpt enough oxygen--> brain cell death, so the host will death also..mati pun host sekor nie...ntah betul ke tak tau aku tulis nie. malas da nak rujuk buku..nanti jer la.. (",)..
end of yesterday. really need problem for today... :-(
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| atleast this dog have something to when they get bored. |
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